Why in OBQ06 explanation c 8 radiculopathy state all dip flexion of all fingers affected while in the article mention c8 radiculopathy only dip flexion of index and middle finger affected plz clarify thanx
A 52-year-old woman underwent a C5/6 ACDF for cervical radiculopathy through a left-sided approach two years ago. Radiographs are shown in Figure A. She has had an altered voice since this operation. Recently, the patient has developed myelopathic symptoms including gait instability and dexterity problems with her hands. An MRI shows a fusion at C5/6, and an adjacent-level midline disc herniation at C4/5 with cord compression and myelomalacia. Laryngoscopy of the vocal cords demonstrates abnormal function of the vocal cords on the left hand side. Which of the following is the most appropriate treatment for this patient?
Physical therapy and NSAIDS
High dose methylprednisone
C5/6 hardware removal and C4/5 ACDF using a left sided anterior approach
C5/6 hardware removal and C4/5 ACDF using a right sided anterior approach
C5 to C7 posterior laminectomy and fusion
Very informative !!!! Thanx
A 38-year-old male presents with a cervical disc herniation at the C7/T1 level with associated foraminal stenosis, but no significant central stenosis. What would be the expected symptoms and physical exam findings.
Numbness of the lateral shoulder and deltoid weakness
Numbness of 2nd and 3rd fingers and triceps weakness
Numbness of the thumb with weakness to wrist extension
Numbness of 5th finger with weakness to long flexor function in all digits and thumb
Numbness of the medial elbow and weakness to long finger flexion of the 4th and 5th digits only
The finger flexion will be effected by c8 radiculopathy and high ulnar nerve neuropathy( cubital tunnel)...on other hand flexion of dip little and ring finger will be only effected and sparing dip of index and middle in lower ulnar nerve peripheropathy(fdp innervation)
Postoperative radiculopathy is a known complication of posterior cervical decompression for myelopathy. One potential mechanism of nerve root injury is thought to be tethering of the nerve root with dorsal migration of the spinal cord. What is the most common radicular pattern seen with this condition?
Motor-dominant radiculopathy with weakness of the deltoid
Sensory-dominant radiculopathy with pain in the lateral shoulder
Motor-dominant radiculopathy with weakness of the wrist extensors
Sensory-dominant radiculopathy with pain in the lateral forearm
Motor-dominant radiculopathy with weakness of the triceps
Does not posterior deco produce kyphotic alignement and therefore the spinal cord will be pushed ventrally??
The treatment section under PLC has some discrepancies. The first scenario should be nonoperative, but it says the patient should be treated with surgery. And in the operative section, the total score should be 5, not 2 (it should also say score > 4 = operative).
A 37-year-old male feels a "pop" in his low back while lifting a heavy object. Four weeks later he continues to have significant low back pain, with no complaints of symptoms in his leg. A T2-weighted MRI is shown in Figure A with a red arrow identifying abnormal signal intensity in an anatomic structure. What type of collagen is primarily responsible for the biological properties of this anatomic structure?
"An anatomic structure", the annulus. "This anatomic structure", the nuclear material? Why make the question so difficult to interpret? How about " What is the black structure in which the white spot is located?"
I believe there is an error in the Prognosis section. In the current list of risk of progression by morphology, incarcerated hemivertebrae are listed as having a higher risk of curve progression than unincarcerated hemivertebrae. For the reasons expressed in the article cited by Dr. Shilt, segments above and below accomodate incarcerated hemivertebrae but not unincarcerated hemivertebrae.
In patients with incomplete spinal cord injuries what is the most important prognostic variable relating to neurologic recovery?
Severity of neurologic deficit
Mechanism of injury
Administration of spinal dose steroids within 8 hours
Early definitive surgery
Dr Derek moore..i think the severity of the injury was indentified by complete vs incomplete...now what is remaining the mechanism of incomplete injury that is subdivided into central cord syndrome(caused by HYPEREXTENSION) which carries agood prognosis..and anterior cord syndrome( caused by HYPERFLEXION) which carries a bad prognosis roughly speaking...so answer 2 wouldn't be the right answer plz clarify.. thanx in advance....