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Review Question - QID 213112

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QID 213112 (Type "213112" in App Search)
A 56-year-old woman sustains the closed injury depicted in Figures A-B. On examination, her wrist is mildly swollen and she is unable to actively oppose her thumb. She also complains of some paresthesias in her thumb and index finger. The patient undergoes closed reduction and splinting; however, her paresthesias worsen significantly in the next 12 hours. What is the likely mechanism of her paresthesias and what is the most appropriate treatment?
  • A
  • B

Nerve compression; open reduction internal fixation with open carpal tunnel release

95%

2033/2133

Nerve laceration; open reduction internal fixation with primary nerve repair or grafting

1%

20/2133

Decreased arterial inflow; fasciotomy with open reduction internal fixation

1%

20/2133

Reflex sympathetic dystrophy; vitamin C

0%

8/2133

Nerve compression; repeat closed reduction

1%

21/2133

  • A
  • B

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This patient is presenting with signs of acute carpal tunnel syndrome (CTS) in the setting of a displaced distal radial fracture. The pathogenesis of acute CTS is nerve compression, requiring urgent open carpal release with open reduction internal fixation (ORIF).

Acute CTS is a well-recognized phenomenon after distal radial fractures. Risk factors include ipsilateral upper extremity fractures, translation of the fracture fragments, and articular distal radius fractures (DRFs). Acute CTS can manifest with paresthesias in the median nerve distribution and opponens pollicis weakness. Acute CTS is an indication for urgent surgical decompression of the median nerve.

Odumala et al. performed a study to evaluate the role of carpal tunnel decompression in the prevention of median nerve dysfunction after buttress plating of DRFs. They reported that prophylactic decompression of the carpal tunnel results in twice the relative odds of developing median nerve dysfunction, which routinely self-resolved. They concluded that prophylactic median nerve decompression does not alter the course of median nerve dysfunction and may actually increase postoperative morbidity.

Medici et al. performed a case-control study to investigate whether carpal tunnel release (CTR) during fixation DRFs improves outcomes. They reported no statistically significant difference between the groups in VAS and Mayo Wrist Scores, however, an increased risk of subsequent CTR in the group who underwent ORIF with no CTR at the index procedure. They concluded that the release of the transverse carpal ligament during ORIF may reduce the incidence of postoperative median nerve dysfunction.

Niver et al. reviewed CTS after DRFs. They reported that acute CTS noted at the time of DRF warrants urgent surgical release of the carpal tunnel and fracture fixation, and that delayed CTS presenting after a distal radius fracture has healed may be managed in the standard fashion for CTR. They concluded that there is no role for prophylactic CTR at the time of distal radius fixation in a patient who is asymptomatic.

Figures A and B depict a displaced apex volar DRF and a mildly displaced ulnar styloid fracture.

Incorrect Answers:
Answer 2: Acute CTS after DRF most often commonly occurs after median nerve compression and contusion, not laceration.
Answer 3: This describes compartment syndrome, which is less likely than acute CTS given the clinical exam described.
Answer 4: Reflex sympathetic dystrophy usually occurs after the acute phase of the DRF.
Answer 5: Acute CTS after DRF requires urgent open carpal tunnel release.



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