Despite recent research developments, Duchenne muscular dystrophy remains a fatal neuromuscular disease, affecting two to three boys in 10,000. It is an inherited X-linked recessive condition caused by a frameshift mutation in the dystrophin gene at the Xp21.2 locus of the X chromosome1 . Dystrophin is a large cell-membrane protein involved in calcium transport in the muscle cell. Boys with Duchenne muscular dystrophy have an absolute absence of dystrophin, leading to deterioration of the muscle cells and replacement with fibrofatty tissue2. This is in contrast to Becker muscular dystrophy, in which less disruptive mutations that do not result in a frame shift lead to production of variable amounts of a smaller, genetically abnormal dystrophin